Publication Date: 04 Sep 2009
Type: Short Commentary
Journal: Journal of Cell Death
Citation: Journal of Cell Death 2009:2 41-44
Pathogenesis of anthrax lethal toxin (LT) is attributed to its ability to cause death of infected cells. New work has demonstrated that increase of mitochondrial F1F0 ATPase activity and subsequent depletion of cellular ATP level are critical early events during LT-induced cell death.
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